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     Contrary to what the “don’t confuse me the facts” media would have us believe, the hormonal etiology of MPB unfortunately involves much more than dihydrotestosterone, (DHT).  Proscar/Propecia/Avodart , though moderately and transiently helpful for some who use it as a sole treatment, offer little hope for a cosmetically significant sustainable result.  This may actually be good news though.

     There have been other enzymes and hormones that have been shown to play a role in the development and progression of MPB that are likely at least as significant as DHT.  If these are addressed in the treatment process than the results one might expect would be significantly better.

     The good news in all this is that there are ways to neutralize and address these enzymes and hormones in ways that are actually health promoting.

     In a previous update we went into detail about the importance of inhibiting aromatase for overall health and hair growth, and *especially* when using a 5alpha reductase inhibitor, like Proscar/Propecia/Avodart.  We also outlined several ways to accomplish this using various natural and pharmaceutical compounds.

     In addition to DHT and aromatase, there is data strongly suggesting that Androstendione , Prolactin and specific estrogen metabolites play deleterious roles in both men’s and women’s hair loss.

     In this update we will give a succinct and simplistic overview of the roles Androstendione and Prolactin in the pathogenesis of MPB and discuss a newly formulated multicomponent phytosterol compound that specifically addresses these factors.

     In our next update we will further delve into role of specific estrogen metabolites and additional modes by which to target excessive DHT production that are addressed by this compound as well.

     Dr. Peter Proctor M.D., PhD, holder of several hair loss treatment patents, has maintained for years that Androstendione is at least as significant as DHT in hair follicle regression.

     Androstendione is endogenously created from testosterone via an isozyme known as 17 beta hydroxysteroid dehydrogenase, (17BHSD).  “Andro” is it is called, was sold as a prohormone in the 90’s and was designed to increase muscle anabolism.  All “Andro” ended up increasing was hair fall out, estrogen levels, and acne.

     Androstendione can fortunately be reduced by inhibiting 17BHSD.  How???

     17BHSD inhibition can fortunately be readily accomplished by increasing the endogenous production of the lignan, Enterolactone.  Enterolactone is produced by select dietary precursors.














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